Beta-actin variant is necessary for Enterovirus 71 replication

Yan Long Edmund Lui, Zhiyang Lin, Jia Jun Lee, Vincent Tak Kwong Chow, Chit Laa Poh, Eng Lee Tan*

*Corresponding author for this work

    Research output: Contribution to journalArticlepeer-review

    10 Citations (Scopus)

    Abstract

    Enterovirus 71 (EV71) is one of the main etiological agents of the Hand, Foot and Mouth Disease (HFMD) and has been known to cause fatal neurological complications such as herpangina, aseptic meningitis, poliomyelitis-like paralysis and encephalitis. EV71 is endemic in the Asia-Pacific region and causes occasional epidemics. In order to better understand EV71 infection, we compared the proteome between EV71-susceptible and EV71-resistant human Rhabdomyosarcoma (RD) cell line. We found significant differences in the β-actin variants between the EV71-susceptible RD cells and EV71-resistant RD cells, suggesting that β-actin, in association with other proteins such as annexin 2 is required in vesicular transport of EV71. This finding further support our previous study that actin potentially plays a role in pathogenesis and the establishment of the disease in HFMD.

    Original languageEnglish
    Pages (from-to)607-610
    Number of pages4
    JournalBiochemical and Biophysical Research Communications
    Volume433
    Issue number4
    DOIs
    Publication statusPublished - 19 Apr 2013

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