CTLA-4 and multiple sclerosis: The A49G single nucleotide polymorphism shows no association with multiple sclerosis in a Southern Australian population

Bradley N. Wray, Jim Stankovich, Lucy Whittock, Terence Dwyer, Anne Louise Ponsonby, Ingrid A.F. van der Mei, Bruce Taylor, Joanne Dickinson, Simon Foote, Brendan J. McMorran*

*Corresponding author for this work

Research output: Contribution to journalArticlepeer-review

11 Citations (Scopus)

Abstract

Multiple sclerosis (MS) is a chronic autoimmune disorder that causes inflammatory demyelination and axonal damage in the central nervous system (CNS). We have investigated whether the A49G single nucleotide polymorphism (SNP) genotype of the CTLA-4 gene influenced the development of MS in Southern Australians as well as the interaction of this SNP with the DRB1*15 haplotype. There were no significant (P < 0.05) associations between the A49G genotype and risk of MS, either before or after stratification for presence of the DR15 haplotype.

Original languageEnglish
Pages (from-to)139-142
Number of pages4
JournalJournal of Neuroimmunology
Volume196
Issue number1-2
DOIs
Publication statusPublished - 30 May 2008
Externally publishedYes

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