Oxidative stress, intrauterine growth restriction, and developmental programming of type 2 diabetes

Cetewayo S. Rashid; Amita Bansal; Rebecca A. Simmons

doi:10.1152/physiol.00023.2018

Oxidative stress, intrauterine growth restriction, and developmental programming of type 2 diabetes

Cetewayo S. Rashid, Amita Bansal, Rebecca A. Simmons^*

^*Corresponding author for this work

Research output: Contribution to journal › Article › peer-review

61 Citations (Scopus)

Abstract

Intrauterine growth restriction (IUGR) leads to reduced birth weight and the development of metabolic diseases such as Type 2 diabetes in adulthood. Mitochondria dysfunction and oxidative stress are commonly found in key tissues (pancreatic islets, liver, and skeletal muscle) of IUGR individuals. In this review, we explore the role of oxidative stress in IUGR-associated diabetes etiology.

Original language	English
Pages (from-to)	348-359
Number of pages	12
Journal	Physiology
Volume	33
Issue number	5
DOIs	https://doi.org/10.1152/physiol.00023.2018
Publication status	Published - 2018
Externally published	Yes

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title = "Oxidative stress, intrauterine growth restriction, and developmental programming of type 2 diabetes",

abstract = "Intrauterine growth restriction (IUGR) leads to reduced birth weight and the development of metabolic diseases such as Type 2 diabetes in adulthood. Mitochondria dysfunction and oxidative stress are commonly found in key tissues (pancreatic islets, liver, and skeletal muscle) of IUGR individuals. In this review, we explore the role of oxidative stress in IUGR-associated diabetes etiology.",

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AB - Intrauterine growth restriction (IUGR) leads to reduced birth weight and the development of metabolic diseases such as Type 2 diabetes in adulthood. Mitochondria dysfunction and oxidative stress are commonly found in key tissues (pancreatic islets, liver, and skeletal muscle) of IUGR individuals. In this review, we explore the role of oxidative stress in IUGR-associated diabetes etiology.

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JO - Physiology

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Oxidative stress, intrauterine growth restriction, and developmental programming of type 2 diabetes

Abstract

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