Signal transducer and activator of transcription 3 (STAT3) mutations underlying autosomal dominant hyper-IgE syndrome impair human CD8⁺ T-cell memory formation and function

Megan L. Ives, Cindy S. Ma, Umaimainthan Palendira, Anna Chan, Jacinta Bustamante, Stephanie Boisson-Dupuis, Peter D. Arkwright, Dan Engelhard, Diana Averbuch, Klaus Magdorf, Joachim Roesler, Jane Peake, Melanie Wong, Stephen Adelstein, Sharon Choo, Joanne M. Smart, Martyn A. French, David A. Fulcher, Matthew C. Cook, Capucine PicardAnne Durandy, Miyuki Tsumura, Masao Kobayashi, Gulbu Uzel, Jean Laurent Casanova, Stuart G. Tangye, Elissa K. Deenick^*

^*Corresponding author for this work

Research output: Contribution to journal › Article › peer-review

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Signal transducer and activator of transcription 3 (STAT3) mutations underlying autosomal dominant hyper-IgE syndrome impair human CD8⁺ T-cell memory formation and function

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Immunology and Microbiology

Signal transducer and activator of transcription 3 (STAT3) mutations underlying autosomal dominant hyper-IgE syndrome impair human CD8+ T-cell memory formation and function

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Immunology and Microbiology

Signal transducer and activator of transcription 3 (STAT3) mutations underlying autosomal dominant hyper-IgE syndrome impair human CD8⁺ T-cell memory formation and function